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Fighting the epidemic of pediatric metabolic dysfunction-associated steatotic liver disease: Role of non-invasive diagnostics and early pharmacological intervention - PubMed

Fighting the epidemic of pediatric metabolic dysfunction-associated steatotic liver disease: Role of non-invasive diagnostics and early pharmacological intervention - PubMed

Source : https://pubmed.ncbi.nlm.nih.gov/41640966/

The global rise in childhood obesity has made metabolic dysfunction-associated steatotic liver disease (MASLD) the leading cause of pediatric liver disease. Studies have consistently reported alarmingly high rates of advanced...

Mini-review addresses rising pediatric MASLD, emphasizing non-invasive diagnostics for early fibrosis detection and advocating early pharmacological intervention, including GLP-1 receptor agonists, to prevent progression and improve long-term outcomes in children.

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Did you know? Hormonal imbalances can subtly influence lipid metabolism, insulin sensitivity, body weight, and renal function. Disruptions in calcium, vitamin D, and magnesium signaling may amplify cardiometabolic risk, underscoring the need for integrated endocrine monitoring across obesity, diabetes, and lipid disorders to support long-term metabolic health.

Could a more holistic approach to hormonal monitoring improve cardiometabolic outcomes across diverse metabolic conditions?

NCCN Guidelines

Could a more holistic approach to hormonal monitoring improve cardiometabolic outcomes across diverse metabolic conditions?

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Emerging evidence links hormonal imbalance to metabolic dysfunction, oxidative stress, and elevated cardiovascular risk. Conditions marked by insulin resistance, dyslipidemia, and obesity share disrupted endocrine signaling that accelerates cardiometabolic complications across populations.

Explore endocrine–metabolic connections

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Pathogenic analysis of post-transplantation obesity: A comprehensive systematic review - PubMed

Pathogenic analysis of post-transplantation obesity: A comprehensive systematic review - PubMed

Source : https://pubmed.ncbi.nlm.nih.gov/41607827/

Post-transplant obesity arises from a complex interplay of pharmacological, behavioral, and molecular factors. A multidisciplinary approach-incorporating pharmacological modification, nutritional management, physical activity, and molecular-targeted therapies-is essential to mitigate obesity and...

Systematic review identifies pharmacological, behavioral, and molecular drivers of post-transplant obesity, highlighting immunosuppressant effects, lifestyle factors, and adipokine dysregulation, and advocating multidisciplinary strategies to improve long-term transplant outcomes.

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