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What is the exact mechanism which results in the clinical outcomes, and Heart Failure patients with SGPT2 inhibitors?

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  • 2yr
    Thanks so much for this intriguing question.

    Numerous mechanisms likely underlie the cardioprotective effects of SGLT2. The following excerpt from JACC: Basic to Translational Science outlines them as follows:

    “These include beneficial effects of SGLT2 inhibition on the following: 1) blood pressure lowering; 2) increasing diuresis/natriuresis; 3) improving cardiac energy metabolism; 4) preventing inflammation; 5) weight loss; 6) improving glucose control; 7) inhibiting the sympathetic nervous system; 8) preventing adverse cardiac remodeling; 9) preventing ischemia/reperfusion injury; 10) inhibiting the cardiac Na+/H+ exchanger; 11) inhibiting SGLT1; 12) reducing hyperuricemia; 13) increasing autophagy and lysosomal degradation; 14) decreasing epicardial fat mass; 15) increasing erythropoietin (EPO) levels; 16) increasing circulating provascular progenitor cells; 17) decreasing oxidative stress; and 18) improving vascular function.”

    If interested, please refer to the rest of the article for a more detailed analysis:
  • Mechanisms of Cardiovascular Benefits of Sodium Glucose Co-Transporter 2 (SGLT2) Inhibitors: A State-of-the-Art Review

    Mechanisms of Cardiovascular Benefits of Sodium Glucose Co-Transporter 2 (SGLT2) Inhibitors: A State-of-the-Art Review

    Source : https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7315190/

    Recent clinical trials have shown that sodium glucose co-transport 2 (SGLT2) inhibitors have dramatic beneficial cardiovascular outcomes. These include a reduced incidence of cardiovascular death and heart failure hospitalization in people with and without ...

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